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One possible explanation is that low levels of caspase-1 activity are sufficient to initiate a cascade leading to lytic death, and once gasdermin D is cleaved, downstream pore formation proceeds independently of caspase activity
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Thus, these conserved interactions promote the formation and stability of the bypass motif, which is a key structural determinant for the high potency of Amy analogues at AMYRs 15,16
The Takeaway Mitochondrial dysfunction and undermethylation are two sides of the same coin
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